Quantitative analysis of cardiac lesions in chronic canine chagasic cardiomyopathy

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MetadadosDescriçãoIdioma
Autor(es): dc.creatorCaliari, Marcelo Vidigal-
Autor(es): dc.creatorMachado, Raquel do Pilar-
Autor(es): dc.creatorLana, Marta de-
Autor(es): dc.creatorCajá, Rosângela Aparecida França-
Autor(es): dc.creatorCarneiro, Cláudia Martins-
Autor(es): dc.creatorBahia, Maria Terezinha-
Autor(es): dc.creatorSantos, César Augusto Bueno dos-
Autor(es): dc.creatorMagalhães, Gustavo Albergaria-
Autor(es): dc.creatorSampaio, Ivan Barbosa Machado-
Autor(es): dc.creatorTafuri, Washington Luiz-
Data de aceite: dc.date.accessioned2019-11-06T13:25:01Z-
Data de disponibilização: dc.date.available2019-11-06T13:25:01Z-
Data de envio: dc.date.issued2012-09-21-
Data de envio: dc.date.issued2012-09-21-
Data de envio: dc.date.issued2002-
Fonte completa do material: dc.identifierhttp://hdl.handle.net/123456789/1416-
Fonte: dc.identifier.urihttp://educapes.capes.gov.br/handle/capes/554934-
Descrição: dc.descriptionLesions observed in chronic chagasic cardiopathy frequently produce electrocardiographic alterations and affect cardiac function. Through a computerized morphometrical analysis we quantified the areas occupied by cardiac muscle, connective and adipose tissues in the right atrium of dogs experimentally infected with Trypanosoma cruzi. All of the infected dogs showed chronic myocarditis with variable reduction levels of cardiac muscle, fibrosis and adipose tissue replacement. In the atrial myocardium of dogs infected with Be78 and Be62 cardiac muscle represented 34 and 50%, fibrosis 28 and 32% and adipose tissue 38 and 18%, respectively. The fibrosis observed was both diffuse and focal and mostly intrafascicular, either partially or completely interrupting the path of muscle bundles. Such histological alterations probably contributed to the appearance of electrocardiographic disturbances verified in 10 out 11 dogs which are also common in human chronic chagasic cardiopathy. Fibrosis was the most important microscopic occurrence found since it produces rearrangements of collagen fibers in relation to myocardiocytes which causes changes in anatomical physiognomy and mechanical behavior of the myocardium. These abnormalities can contribute to the appearance of cardiac malfunction, arrhythmias and congestive cardiac insufficiency as observed in two of the analyzed dogs. Strain Be78 caused destruction of atrial cardiac muscle higher than that induced by strain Be62.-
Idioma: dc.languageen-
Direitos: dc.rightsA Revista do Instituto de Medicina Tropical de São Paulo, permite que o Repositório Institucional da Universidade Federal de Ouro Preto deposite uma cópia eletrônica dos artigos publicados por esse periódico em que ao menos um dos autores faça parte da comunidade cientifica da Universidade Federal de Ouro Preto. Fonte: Licença concedida pelo Periódico: enviado no dia 16/12/2013.-
Palavras-chave: dc.subjectMyocarditis-
Palavras-chave: dc.subjectCongestive cardiac insufficiency-
Palavras-chave: dc.subjectFibrosis-
Palavras-chave: dc.subjectTrypanosoma cruzi-
Palavras-chave: dc.subjectChagasic cardiomyopathy-
Título: dc.titleQuantitative analysis of cardiac lesions in chronic canine chagasic cardiomyopathy-
Aparece nas coleções:Repositório Institucional - UFOP

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